Causes, Identification and Repair of loss of Common Ground in coordination in ATM (Air Traffic Management)

Over a century has passed since humans took to commercial flying. Traditional safety practices have worked well but the last decade has seen the need for an updated understanding of ATM safety. The modern safety views are complementary to the traditional ones but are also a new way of understanding and enabling safety practices. This master thesis report presents a comprehensive review of the sources chosen from literature to better understand how a complex sociotechnical system, such as ATM, would operate. Certain selected coordination aspects will be the focus of this master thesis and will be used to model and analyse an ATM case. The ultimate aim of this research project is to add to the growing body of knowledge in the field of ATM safety, to make flying increasingly safer and to enable a complex system to be resilient.Aerospace EngineeringAir Transport Operation

Logistic regression model (<i>R²</i> = 0.12) to predict OM proneness in 653 children.

<p>Only statistically significant results are shown above at P value <0.05.</p>a<p>The inference for the factors of race, IL-1β (−511), and IL-10 (−1082) is a based on a joint hypothesis; thus the degrees of freedom on the chi-square (11.91) is 3, as opposed to 1 on the other inferences.</p>b<p>Wild type genotype.</p>c<p>Either hetero- or homozygous polymorphic genotype.</p>d<p>Homozygous polymorphic genotype.</p

Demographic and clinical characteristics of 653 study children.

a<p>Whites of non-Hispanic ethnicity.</p>b<p>Any duration of breast feeding.</p>c<p>Any duration of exposure to cigarette smoke.</p>d<p>OM susceptibility in immediate family members.</p

Allele and genotype frequencies among 653 study subjects.

<p>RS number = Reference single nucleotide polymorphism (SNP) number.</p><p>CCR5 = C-C chemokine receptor type 5, CX3CR1 = CX3C chemokine receptor 1, ICAM1 = Inter-Cellular Adhesion Molecule 1, IL-1β = Interleukin 1β, IL-2 = Interleukin 2, IL-5 = Interleukin 5, IL-6 = Interleukin 6, IL-8 = Interleukin 8, IL-10 Interleukin 10, IL-12 = Interleukin 12, IL13 = Interleukin 13, IL-18 = Interleukin 18, MBL = Mannose-binding lectin, TGF-β1 = Transforming growth factor β, TLR4 = Toll-like receptor, TNFα = Tumor necrosis factor α.</p

Additional file 1: of Testing the effects of narrative and play on physical activity among breast cancer survivors using mobile apps: study protocol for a randomized controlled trial

CONSORT 2010 checklist of information to include when reporting a randomised trial. (DOC 218 kb

Additional file 1: of The feasibility and RE-AIM evaluation of the TAME health pilot study

Five A’s Counseling Script. (DOCX 27 kb

Cell and cytokine profile of neutrophil-high (Neu-High) asthma and neutrophil-normal (Neu-Normal) asthma.

<p>The upper limit of percent of neutrophils in the BAL fluid of healthy subjects was 2.4%. We separated all subjects with asthma into neutrophil-high (neutrophils% > 2.4%), and neutrophil-normal (neutrophil≤2.4%) groups. Compared to Neu-Normal asthma, Neu-High asthma had higher IL-8 levels (p<0.01) and lower % predicted FEV₁ (p<0.01), but similar levels of eosinophil %, IL-5, IL-13, IL-16, and PDGF-bb. Data are expressed as means ± SEM. *<i>P</i> < .05, **<i>P</i> < .01.</p

Patient characteristics.

<p>Results expressed in means and range.</p><p>*Statistical significant compared to healthy group</p><p>⁺Statistical significant compared to controlled asthma</p><p>Patient characteristics.</p

Cell and cytokine profile of eosinophil-high (Eos-High) asthma and eosinophil-normal (Eos-Normal) asthma.

<p>The upper limit of percent of eosinophils in the BAL fluid of healthy subjects was 0.3%. We separated all subjects with asthma into either eosinophil-high (eosinophils > 0.3%) and eosinophil-normal (eosinophils≤0.3%) groups. Compared to Eos-Normal asthma, Eos-High asthma had higher levels of IL-5 (p<0.05), IL-13 (p<0.05), IL-16 (p<0.05), and PDGF-bb (p<0.05), but same % neutrophils, IL-8, and FEV₁. Data are expressed as means ± SEM. *<i>P</i> < .05, **<i>P</i> < .01.</p

Correlation of FEV1 to eosinophil, neutrophil, IL-5 and IL-8 levels in asthma.

<p>(A) Percentages of neutrophils and concentrations of IL-8 in BAL fluids of subjects with controlled asthma and uncontrolled asthma. (B) Correlations of concentrations of IL-8 with the percentages of neutrophils in the BAL fluid from all subjects with asthma (left panel). Correlation of percentages of neutrophils and concentrations of IL-8 in BAL fluid with percent predicted FEV₁ (middle and right panels, respectively). (C) Correlation of concentrations of IL-5 with the percentages of eosinophil in the BAL fluid from all subjects with asthma (left panel). Correlation of percentages of eosinophils and concentrations of IL-5 in BAL fluid with percent predicted FEV₁ (middle and right panels, respectively). Data are expressed as means ± SEM. * = <i>P</i> < .05, ** = <i>P</i> < .01, *** = <i>P</i> < .001, ***<i>* = P</i> < .0001.</p